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Etorphine HCL (M99) 10ml – 10mg/ml, 99.9% Purity

$105.00 $85.00

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Etorphine HCL (M99)

Etorphine HCL (M99) is a semi-synthetic opioid possessing an analgesic potency approximately 1,000–3,000 times that of morphine. It was first prepared in 1960 from oripavine, which does not generally occur in opium poppy extract but rather the related plants Papaver orientale and Papaver bracteatum. It was later reproduced in 1963 by a research group at MacFarlan Smith in Gorgie, Edinburgh, led by Kenneth Bentley. It can also be produced from thebaine.

Etorphine HCl (M99, 10 mg/mL, is considered the most widely used UPO in zoo and wildlife anesthesia,5 and is the induction agent of choice for elephant, rhinoceros, nondomestic equids, and other hoofstock. It is often combined with azaperone, medetomidine, midazolam, or azaperone to produce muscle relaxation.6 The availability of etorphine since its first use and description in the late 1960s revolutionized the ability of veterinarians to safely capture and restrain many species that previously could not be handled.

Worldwide, etorphine hydrochloride (M99) is the most common opioid agent used for immobilization of hoofstock. Etorphine may be used either alone or in combination with neuroleptic synergistic agents. Induction times with etorphine are usually longer compared with other opioid agents, such as carfentanil. The most common side effect associated with etorphine immobilization is pronounced respiratory depression, but common opioid effects (e.g., excitement, poor muscle relaxation, bradycardia or tachycardia, renarcotization) may be seen after administration of the opioid antagonist.

Worldwide, etorphine hydrochloride (M99) is the most common opioid agent used for immobilization of hoofstock. Etorphine may be used either alone or in combination with neuroleptic synergistic agents. Induction times with etorphine are usually longer compared with other opioid agents, such as carfentanil. The most common side effect associated with etorphine immobilization is pronounced respiratory depression, but common opioid effects (e.g., excitement, poor muscle relaxation, bradycardia or tachycardia, renarcotization) may be seen after administration of the opioid antagonist.

 

 

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